Cholinergic neurodegeneration and cholesterol metabolism dysregulation by constitutive p75^NTR signaling in the p75^exonIII-KO mice.

SAMP8-p75 exonIII−/− 49.5 ± 0.3 μm N = 4), however, quantification of the number of BrdU + cells showed that in the SAMP8-p75 exonIII−/− mice at the age of 2 months there was a slight decrease in the proliferative activity of the neural stem cell niche that did not reach statistical significance ( Figures 1F – I ).

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