The identification of BCL-XL and MCL-1 as key anti-apoptotic proteins in medulloblastoma that mediate distinct roles in chemotherapy resistance.

5E, F ), and between BAX and BCL-XL in ONS76 cells, suggesting a potential resistance mechanism whereby BAX/BAK are sequestered by BCL-XL upon treatment with cisplatin, though these increases did not reach statistical significance (Fig. 5G, H ).

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