The other proteins also showed a clear trend of decrease, including AOX1, MYLK3 (myosin light chain kinase 3), and STK38L (serine/threonine-protein kinase 38-like), while β-actin had no change, and the total protein loading was equalized ( Figure 6 B and Figure S4C ).
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Cardiomyocyte-Specific Loss of Glutamyl-prolyl-tRNA Synthetase Leads to Disturbed Protein Homeostasis and Dilated Cardiomyopathy.
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