Inhibition of CSF-1R and IL-6R prevents conversion of cDC2s into immune incompetent tumor-induced DC3s boosting DC-driven therapy potential.

CD1c + CD14 − and CD1c + CD14 + cells induced comparable amounts of autologous antigen-specific CD4 T cell proliferation and slightly lower CD8 T cell proliferation, which did not reach statistical significance ( Figures S2 A and S2B).

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