5 a, S9a), and consistent with the two-factor analysis, their induction was repressed in ALK1 -mutated HMVECs, except for SLC6A6 , whose difference between CTL and MUT HMVECs did not reach statistical significance (Fig.
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Impact of heterozygous ALK1 mutations on the transcriptomic response to BMP9 and BMP10 in endothelial cells from hereditary hemorrhagic telangiectasia and pulmonary arterial hypertension donors.
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