The redox-active defensive Selenoprotein T as a novel stress sensor protein playing a key role in the pathophysiology of heart failure.

Although we observed a tendency of an increase of desmin levels in the cardiac insoluble fraction of SHHF group compared with that of WST group, as well as in that of SHHF + PSELT group with respect to that of SHHF group, these differences did not reach statistical significance (Fig. 2 B).

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