Hydrogen therapy promotes macrophage polarization to the M2 subtype in radiation lung injury by inhibiting the NF-κB signalling pathway.

Treatment group 1 showed a trend of remission relative to the model group, but it was not statistically significant, and the recovery of the mice's pulmonary function might be delayed relative to the pathological changes.These results indicate that hydrogen can significantly improve lung function in mice ( Table 1 ). 3.2 Hydrogen therapy alleviated pathological changes in lung tissue in mice with radiation lung injury The pathology of radiation lung injury is characterised by diffuse alveolitis, pathological proliferation of fibroblasts, and deposition of extracellular matrix in the interstitium and alveolar spaces.

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