In addition, allelic copy number loss also coincided with reduced expression of USP15 and SCAF1 and patients with deep or shallow USP15 or SCAF1 deletions showed a significant trend towards a shorter overall survival (Fig. 6b and Supplementary Fig. 8a ).
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In vivo CRISPR screens reveal SCAF1 and USP15 as drivers of pancreatic cancer.
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