TRIM59 deficiency promotes M1 macrophage activation and inhibits colorectal cancer through the STAT1 signaling pathway.

Although the tumor weight did not reach statistical significance ( p = 0.0632), there was a noticeable reduction in tumor burden in TRIM59 f/f Lyz2-cre mice (Fig. 6 C).

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Although there were no statistically significant differences in the levels of MCP-1 , IL-1β , CXCL9 , CXCL10 , and CXCL11 , an increasing trend was observed in TRIM59 f/f Lyz2-cre mice (Fig. 2 C).

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