Moreover, the immunosuppressive marker IL10 and the pro-fibrotic Tissue Inhibitor of Metalloproteinases (TIMP1) showed a trend of being downregulated by Dex and Dex MPs in most environments, while LRP1, a marker involved in collagen degradation in pulmonary fibrosis [ 11 ], was upregulated, although these trends differed based on the external stimuli.
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Biomaterial-mediated intracellular control of macrophages for cell therapy in pro-inflammatory and pro-fibrotic conditions.
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