Monomeric and oligomeric amyloid-β cause distinct Alzheimer's disease pathophysiological characteristics in astrocytes in human glymphatics-on-chip models.

With ARNT as the reference between groups, we showed a trend of lower expression of STMN1 in the oAβ group compared to the control and statistically significantly lower STMN1 expression in the mAβ group compared to the control ( Fig. 3F , statistics in Fig.

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