LCZ696, an angiotensin receptor-neprilysin inhibitor, ameliorates epithelial-mesenchymal transition of peritoneal mesothelial cells and M2 macrophage polarization.

Notably, while the expression levels of fibrosis-associated proteins in the LCZ696-treated group were diminished compared to those in the Valsartan group, this difference did not reach statistical significance ( Figure 2A–F ).

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