Crosstalk between FTH1 and PYCR1 dysregulates proline metabolism and mediates cell growth in KRAS-mutant pancreatic cancer cells.

Kaplan–Meier survival plots suggested a nonsignificant trend toward better survival outcomes with higher miR-5000-3p expression.

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There was a strong trend where the GLS/glutamate-ammonia ligase (GLUL) expression ratio increased with DFX treatment in a dose-dependent manner (Fig. 8d ), but without any statistical significance.

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