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AntimiR treatment corrects myotonic dystrophy primary cell defects across several CTG repeat expansions with a dual mechanism of action.

Sci Adv · 2024 · PMC11463307 · PMID 39383229

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showed a trendno p-value reported
Our observation is in line with previous studies showing lower expression of DMPK expanded alleles ( 47 , 48 ), but DMPK relative expression only showed a trend toward correlating with CTG repeat size ( Fig. 4B ).

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