Deletion of miR-33, a regulator of the ABCA1-APOE pathway, ameliorates neuropathological phenotypes in APP/PS1 mice.

Although there was a trend toward Aβ 42 peptide reduction in the cortex, it did not reach statistical significance (t[9] = 2.057, p = 0.0698, unpaired two‐tailed t test; Figure 2A ).

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