PKC-mediated phosphorylation governs the stability and function of CELF1 as a driver of EMT in breast epithelial cells.

While this increase was mirrored in the context of forced expression of the CELF1 phosphomimetic mutant, forced expression of the CELF1 phosphomutant only marginally increased cell migration as compared to controls, and this increase did not reach statistical significance ( Fig. 3 E ).

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