2 C), and did not reduce the amount of PKAC-interacting Ca V 1.2α 1Δ1821 (the slight increase, 1.47 ± 0.25, n = 8, did not reach statistical significance).
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Tripartite interactions of PKA catalytic subunit and C-terminal domains of cardiac Ca<sup>2+</sup> channel may modulate its β-adrenergic regulation.
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