The phenotypic alterations of the long bones in both conventional and osteoblast/osteocyte-specific Bbx-deficient mice persisted for 8 weeks; however, the changes in some bone parameters did not reach statistical significance, indicating that the abnormal phenotype and parameters of the skeleton were partially ameliorated as growth progressed.
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The transcription factor BBX regulates phosphate homeostasis through the modulation of FGF23.
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