hAMSCs regulate EMT in the progression of experimental pulmonary fibrosis through delivering miR-181a-5p targeting TGFBR1.

3 F), the lung indices of mice in the model group were significantly higher than those of mice in the control group ( P < 0.0001); the lung indices of mice in the hAMSCs and AGO-hAMSCs groups decreased to different degrees compared with those of mice in the model group ( P < 0.05, P < 0.01), and those of mice in the ANTA-hAMSCs group showed a decreasing trend, although not significant, compared with those of mice in the model group.

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E-CAD, N-CAD, and VIMENTIN protein expression in the ANTA-hAMSCs group showed a trend, although not significant, compared with the model group, and the TGFBR1 protein expression level was reduced ( P < 0.05).

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