Targeted inhibition of Gus-expressing <i>Enterococcus faecalis</i> to promote intestinal stem cell and epithelial renovation contributes to the relief of irinotecan chemotoxicity by dehydrodiisoeugenol.

Our findings indicated an increased presence of g_Enterococcus in the fecal samples of colorectal cancer patients undergoing CPT11 chemotherapy compared to both healthy subjects and colorectal cancer patients not receiving CPT11 chemotherapy; however, this increase did not reach statistical significance.

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