In our study, DEX treatment in models of asthmatic mothers showed a trend towards increased ILC2s in the lungs of the offspring, suggesting that maternal glucocorticoid use in asthmatic mothers may enhance ILC2 function in the offspring and increase their risk of asthma.
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Maternal asthma imprints fetal lung ILC2s via glucocorticoid signaling leading to worsened allergic airway inflammation in murine adult offspring.
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