YAP as a potential therapeutic target for myofibroblast formation in asthma.

Although we did not perform an experiment to investigate whether budesonide directly affected YAP activation, we did observe an increasing trend in the expression of TAZ, the homolog of YAP, in budesonide-pretreated cells stimulated with HDM or IL-1β.

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Not only that, TAZ expression tended to increase in the budesonide pretreated groups, although it did not reach statistical significance (Fig. 6 d).

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