Adenine base editing rescues pathogenic phenotypes in tissue engineered vascular model of Hutchinson-Gilford progeria syndrome.

This was matched by an increase in viSMC density in 25:75 edited TEBVs over 3–5 weeks, although the increase in αSMA and MHC11 area in these TEBVs did not reach statistical significance [ Figs. 8(c) , 8(e) , and 8(f) , supplementary material , Fig.

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