Context-specific eQTLs provide deeper insight into causal genes underlying shared genetic architecture of COVID-19 and idiopathic pulmonary fibrosis.

In the remaining three tissues, we found weak evidence of colocalization between the GWAS and ATP11A- eQTL signals (PP4 < 0.408; Table S9 ), likely because of weakly significant eQTL p values in the locus ( p values >10 −4 ).

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