Caspase 3 and caspase 7 promote cytoprotective autophagy and the DNA damage response during non-lethal stress conditions in human breast cancer cells.

While the cleavage-resistant PARP1-DEVA and PARP1-AEVA cells also showed a trend of increased LC3B transcription relative to PARP1-WT ( S5J Fig ), the PARylation and autophagy flux levels showed no differences ( S5 G, S5 I, S5 K, and S5L Fig ).

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