The results showed a trend toward increased Nav1.6 currents (−20 mV) in the OXA+TET1-Control group compared to the vehicle group, whereas TET1 overexpression markedly reduced these currents ( Fig. 6 , K and L , p = 0.0287 vs .
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TET1 participates in oxaliplatin-induced neuropathic pain by regulating microRNA-30b/Nav1.6.
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