ACAA2 Protects Against Cardiac Dysfunction and Lipid Peroxidation in Renal Insufficiency with the Treatment of S-Nitroso-L-Cysteine.

Although there was no significant decrease in the cell viability of si-ACAA2 stimulated by IS, there was a clear trend ( Figure 3 f).

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Interestingly, low concentrations of indoxyl sulfate increased the mRNA level of CPT2 and ETFDH, while only ACADM and ACAA2 showed a decreasing trend, suggesting that ACADM and ACAA2 proteins were more sensitive to stimulation ( Figure 2 a).

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