Enhancing anti-CD3 mAb-mediated diabetes remission in autoimmune diabetes through regulation of dynamin-related protein 1(Drp1)-mediated mitochondrial dynamics in exhausted CD8<sup>+</sup>T-cell subpopulations.

BMC Med · 2025 · PMC11959779 · PMID 40165248

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did not reach statistical significanceno p-value reported

Although the frequency of total exhausted CD8 + T cells was elevated in diabetic mice compared to nondiabetic controls, the difference did not reach statistical significance (Fig. 2 B).

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Enhancing anti-CD3 mAb-mediated diabetes remission in autoimmune diabetes through regulation of dynamin-related protein 1(Drp1)-mediated mitochondrial dynamics in exhausted CD8&lt;sup&gt;+&lt;/sup&gt;T-cell subpopulations.

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Enhancing anti-CD3 mAb-mediated diabetes remission in autoimmune diabetes through regulation of dynamin-related protein 1(Drp1)-mediated mitochondrial dynamics in exhausted CD8<sup>+</sup>T-cell subpopulations.