An observational study revealed that both short-term and long-term O 3 (9.08 ± 4.06 ppb) exposure is linked to glycerophospholipid metabolism, with shorter exposure (12–24 h) positively correlating with PC (phosphatidylcholine) and lysoPC (lyso-phosphatidylcholine) levels, while longer exposure (1–2 weeks) shows a negative trend [ 13 ].
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Revealing the Molecular Mechanisms of Ozone-Induced Pulmonary Inflammatory Injury: Integrated Analysis of Metabolomics and Transcriptomics.
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