A lymphostatin homologue from <i>Chlamydia pecorum</i> inhibits mitogen-activated bovine T cell proliferation and IFNγ production.

While differences relative to ConA-treated cells did not reach statistical significance owing to inter-animal variance, it is biologically significant that the reduction in IFNγ detected was concentration dependent and virtually no IFNγ was detected at concentrations at which lymphocyte proliferation was inhibited.

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