The dichotomous impacts of Semaphorin3E deficiency on exacerbating airway hyperresponsiveness, remodelling, and inflammation in type-2 low and type-2 high asthma models.

As shown in Fig 1D , Sema3E KO mice in the type 2-low asthma model exhibited an increasing trend to sensitivity to methacholine.

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Further, there was also a tendency to higher constrictive responses in the WT type-2 low model compared to Sema3E KO mice, although this difference did not reach statistical significance ( Fig 1C ).

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