At the same time, the level of soluble CXCR4 in urine also showed a decreasing trend in 0 W groups, although the difference did not reach statistical significance (Supplementary Fig. 3 G).
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CXCL12/CXCR4 modulates macrophage efferocytosis to induce glomerular crescent formation and fibrosis via ELMO1/DOCK180/RAC1 signaling in ANCA-associated glomerulonephritis.
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