Inactivation of PI3K-C2α deregulates cell death pathways and sensitizes to endotoxic shock.

Proc Natl Acad Sci U S A · 2025 · PMC12304892 · PMID 40674428

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Since endotoxic shock is dependent on type I interferon signaling ( 36 ) and IFNβ levels showed a trend to be elevated in Pik3c2a flox24-25/D1268A ; Cre ERT2 mice ( SI Appendix , Fig.

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