Neuro-Cells Mitigate Amyloid Plaque Formation and Behavioral Deficits in the APPswe/PS1dE9 Model of Alzheimer Disease While Also Reducing IL-6 Production in Human Monocytes.

Although the latency to exit the lit box was not significantly different ( p = 0.26, Kruskal–Wallis test, Figure 4 H), APP/PS1 mice showed a trend toward longer exit latency, suggesting that anxiety-like behavior was not fully normalized by NC treatment.

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