Upregulating ANKHD1 in PS19 Mice Reduces Tau Phosphorylation and Mitigates Tau Toxicity-Induced Cognitive Deficits.

The levels of PSD95 also show a possible trend of been partially restored by ANKHD1 ( p = 0.058) ( Figure 2 a,d), however such trend was not evident when the male and female mice were analyzed separately.

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Compared to PS19 mice, TauPS19+ ANKHD1 mice exhibited a trend toward reduced GFAP-positive area, though the difference did not reach statistical significance ( p = 0.19 in males and p = 0.064 in females) ( Figure 3 ).

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