Neuroinflammation-mediated YKL-40 correlates with tau pathology and predicts longitudinal cognitive impairment and brain atrophy in Alzheimer's disease, with hypertensive dependency.

As for MRI brain structures, the negative association between baseline levels of CSF YKL-40 and entorhinal cortex volume was at the threshold of significance (β = −0.128, p = 0.049), but did not remain significant following FDR correction.

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