Transcriptomic and functional profiling of <i>Acinetobacter baumannii</i> reveals adaptation to burn patient blood and time-dependent responses to human serum.

While the differences did not reach statistical significance—likely due to limited sensitivity of the instrument causing high variability of the reads between biological replicates— The observed 2.22-fold increase in iron concentration at 8 HPI supports our hypothesis that iron accumulation contributes to the downregulation of acinetobactin genes at this time point.

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