Interestingly, 5‐days post‐MI, in sections from the upper region of the infarct (“A” sections), the abundance of CD206 immunoreactivity in the infarct border region of MI‐MK2 +/+ hearts showed a trend towards being greater than in MI‐MK2 −/− hearts, but the difference did not reach significance.
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MK2 deficiency decreases mortality in male mice during the inflammatory phase after myocardial infarction.
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