In the Gusu in‐house dataset consisting of 24 patients with glioma, TRAF3IP3, TNFAIP3, ICOSLG, IL11, and PTGES, showed a trend of increasing expression with increasing tumor grade, whereas CCL19 was not (Figure S3C , Supporting Information).
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Comprehensive Characterization of the Immune Microenvironment Based on Nested Resampling Machine Learning Framework Identifies TRAF3 Interacting Protein 3 as a Promising Regulator to Improve the Resistance to Immunotherapy in Glioma.
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