Glucosamine mitigates hyperglycemic-induced oxidative stress via the SIRT1 pathway in human corneal epithelial cells.

While the percentage of viable cells was lower in GlcN-treated cell population than in the vehicle control group after exposure to 40 mM glucose, the difference in cell viability between the two groups of HCE-T cells cultured in other designated glucose dosages did not reach statistical significance.

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