However, it was illustrative to identify both NAA38 and LSM3 among the nominally significant dysregulations, given that ATXN2L and both factors mentioned are members of the Lsm protein family and that LSM12 was previously reported as an ATXN2L interactor [ 82 ].
← all excerpts
Conditional ATXN2L-Null in Adult Frontal Cortex CamK2a+ Neurons Does Not Cause Cell Death but Restricts Spontaneous Mobility and Affects the Alternative Splicing Pathway.
1
—
—