IL-1β priming triggers an adaptive stress response that enhances pancreatic β-cell resilience to subsequent cytotoxic inflammatory insult.

7B, C, F ) and showed a trend toward mitigating the CYT-induced increase in the β-cell dedifferentiation marker Aldh1a3 mRNA [ 30 ] (Fig. 7G ).

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IL1-R1 and IL1-R2 mRNA expression levels showed an increasing trend in INS-1E cells pretreated with IL-1β low before CYT challenge (Supplementary Figure 2C, D ).

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