While a trend toward decreased ACAA1 mRNA levels was observed in advanced-stage (III/IV) NPC patients, this association did not reach statistical significance ( Figure 1B , p > 0.05), suggesting that ACAA1 downregulation is a persistent event in NPC pathogenesis.
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Inactivation of Acetyl-CoA Acyltransferase 1 enhances the proliferation and motility of nasopharyngeal carcinoma cells.
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