Factors Released by Polarized Neutrophil-like Cells Modulate Cardiac Fibroblast Phenotype and Limit the Inflammatory Response After Myocardial Infarction.

Although the increase in CCN2 expression did not reach statistical significance in our study, this may reflect the limited sample size rather than a lack of biological relevance, as previous studies have demonstrated that myofibroblast activation assessed by α-SMA expression is significantly impaired in the absence of CCN2 in vitro [ 34 ].

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