Adoptive NK cell transfer confers neuroprotection by attenuating neuroinflammation and alpha-synuclein pathology in a mouse model of synucleinopathy.

One-way ANOVA revealed a trend toward significance (p = 0.069), though not statistically significant ( Fig. 5 b ) .

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Although no significant differences were observed, a decreasing trend in microglial burden (total Iba-1 + area) was detected in the PFF + NK group compared to PFF group, while no significant changes in αSyn burden were observed between PFF groups at 7 weeks p.i.

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Central memory (CD8 CM) and effector memory (CD8 EM) subsets were present at low frequencies across all conditions, though a slight increase in CD8 EM cells was noted in NK-treated animals (up to 1.6% in Mono NK and 1.3% in PFF NK), implying a possible trend toward enhanced memory activation with NK therapy ( Fig. 7 d ) .

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The cortex showed a reduction trend, but it did not reach statistical significance.

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