Indeed, we observed a significant correlation of LAMA5 and ITGA3 with γδTCR blocking and a nearly significant correlation of TGFB1 and γδTCR blocking, but not with any of the other receptors ( online supplemental figure 4E-G ).
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Autologous profiling reveals inter-patient heterogeneity in Vδ2<sup>+</sup>γδTIL responses to glioblastoma driven by extracellular matrix-BTN3A axis.
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