Barely Significant
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Whole-exome sequencing in children with dyslexia implicates rare variants in CLDN3 and ion channel genes.

Hum Genet · 2025 · PMC12738642 · PMID 41442065

1
hedged sentence
0.0300
closest p · 0.6× alpha
0.0300
boldest claim

The sentences

nominally significantP = 0.03actually significant
The CLDN3 variant was nominally significantly associated with dyslexia when comparing the discovery and RD cases with either the TD cohort ( P = 0.03) or the general population (i.e. gnomAD data; P = 0.003) (Supplementary Table 17 ).

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