Although these changes did not reach statistical significance, the results suggest that LVRF has the capacity to enhance angiogenesis in TA muscles, thereby facilitating dystrophin restoration via exon skipping and contributing to amelioration of muscle fiber size in mdx mice.
← all excerpts
Improving angiogenesis ameliorates the efficacy of ASO-based exon skipping for the treatment of Duchenne muscular dystrophy.
1
—
—