Modulation of host inflammatory pathways by <i>Pseudomonas aeruginosa</i> extracellular vesicles in cystic fibrosis: impact of pulmonary exacerbation and elexacaftor-tezacaftor-ivacaftor treatment.

Other infection and inflammatory pathways that were significantly increased or approached significance in cells following P. aeruginosa EV exposure include the role of pattern recognition receptors in recognition of bacteria and viruses, antimicrobial peptides, and neutrophil degranulation ( Table 1 ).

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