Loss of Snhg5 disrupts cell-cycle regulation without altering cystogenesis in a mouse model of polycystic kidney disease.

While G0/G1 and S/G2/M distributions did not reach statistical significance, there was a trend toward decreased G0/G1, and increased S/G2/M compared to control, consistent with impaired late-stage cell-cycle progression in Snhg5 -null cells.

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